Reproduction Abstracts

Introduction: Exposure to an adverse environment around the time of conception can program adult onset disease although mechanisms involved are unclear. Recently we showed that periconceptional alcohol (PC:EtOH) consumption causes placental stress demonstrated by elevated levels of 11βHSD-2, and sex-specific increases in placental glucose transporters and glycogen accumulation. O-linked-N-acetylglucosamine transferase (OGT), an X-linked gene product, res...